“Nicotine inhibits glucose hypermetabolim in aging mice
– ameliorates cellular energy metabolism disorder
-reduces neuroinflammation
– protects from oxidadativ Stress”
Okay, Then I'll get the nicotine patch. What haven't I tried in the last eight years?! Sogar Tenovofir, Immunmodulator, used for HIV, remained ineffective or increased all neuroimmune symptoms.
Naturally, we need hope, where it—- how should I say ——
this disease has a clearly defined clinical appearance: at low(ster) Activity and activity accumulation are increased, severe inflammation is increased and all neuroimmunological symptoms flood the organism.
This pathomechanism, and only this one, it is in my opinion, on which the medical focus must be directed, This is the pathomechanism of the disease Myalgic Encephalomyelitis ( not chronic fatigue syndrome). ME is always new again and again, similar to Long Covid, understood as a post-infectious conclusion. But it is not a post-infectious conclusion, but an independently new clinical picture of the central nervous system, which became established through a post-infectious precursor. I no longer follow ME research, because I'm no longer mentally capable of doing it …. an infection would not behave in this way to activity, although of course you can't be very active with an ongoing infection. But we have long since discovered the differences to Lyme borreliosis, which depicts such a typical ongoing infection and where the aim is to eliminate a pathogen that is actually virulent in the body with antibiotics, Cell detoxification, Dietary supplements, hyperthermia and reactivation etc. This is a post/infection, but myalgic encephalomeylitis is not! It is systemic activation intolerance, which triggers cascades of errors in the brain and body, some for weeks, Months and finally forever thrown into bed. I wish we didn't need these constant cheap hope mongers, because what needs to be stopped and prevented is autoimmunity. Why are we waiting? 1969 for help?
Why?